Ryan A. Altman, Krishna Sharma, Lian G. Rajewski, Paul Chester Toren, Michael J. Baltezor, Mohan Pal, Somnath Narayan Karad
Index: 10.1021/acschemneuro.8b00085
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Opioid peptides are key regulators in cellular and intercellular physiological responses, and could be therapeutically useful for modulating several pathological conditions. Unfortunately, the use of peptide-based agonists to target centrally located opioid receptors is limited by poor physicochemical (PC), distribution, metabolic, and pharmacokinetic (DMPK) properties that restrict penetration across the blood-brain-barrier via passive diffusion. To address these problems, the present manuscript exploits fluorinated peptidomimetics to simultaneously modify PC and DMPK properties, thus facilitating entry into the central nervous system. As an initial example, the present manuscript exploited the Tyr1-ψ[(Z)CF=CH]-Gly2 peptidomimetic to improve PC drug-like characteristics (computational), plasma and microsomal degradation, and systemic and CNS distribution of Leu-enkephalin (Tyr-Gly-Gly-Phe-Leu). Thus, the fluoroalkene replacement transformed an instable in vitro tool compound into a stable and centrally distributed in vivo probe. In contrast, the Tyr1-ψ[CF3CH2-NH]-Gly2 peptidomimetic decreased stability by accelerating proteolysis at the Gly3-Phe4 position.
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