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2-(4-联苯基)-5-苯基恶二唑

2-(4-联苯基)-5-苯基恶二唑用途

2-(4-联苯基)-5-苯基-1,3,4-恶二唑是一种具有高光致发光量子效率的激光染料[1]。

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2-(4-联苯基)-5-苯基恶二唑名称

[ CAS 号 ]:
852-38-0

[ 中文名 ]:
2-(4-联苯基)-5-苯基-1,3,4-噁二唑

[ 英文名 ]:
2-(4-BIPHENYLYL)-5-PHENYL-1,3,4-OXADIAZOLE

[中文别名 ]:

2-(4-联苯基)-5-苯基恶二唑

[英文别名 ]:

EINECS 212-712-0
2-phenyl-5-(4-phenylphenyl)-1,3,4-oxadiazole
MFCD00003100

2-(4-联苯基)-5-苯基恶二唑生物活性

[ 描述 ]:

2-(4-联苯基)-5-苯基-1,3,4-恶二唑是一种具有高光致发光量子效率的激光染料[1]。

[ 相关类别 ]:

研究领域 >> 其他

信号通路 >> 其他 >> 其他

[参考文献]

[1]. Wang C, et al. Polymeric Alkoxy PBD [2-(4-Biphenylyl)-5-Phenyl-1, 3, 4-Oxadiazole] for Light-Emitting Diodes. Advanced Functional Materials, 2001, 11(1): 47-50.

2-(4-联苯基)-5-苯基恶二唑物理化学性质

[ 密度 ]:
1.172g/cm3

[ 沸点 ]:
480.7ºC at 760 mmHg

[ 熔点 ]:
167-169 °C(lit.)

[ 分子式 ]:
C₂₀H₁₄N₂O

[ 分子量 ]:
298.33800

[ 闪点 ]:
240.7ºC

[ 精确质量 ]:
298.11100

[ PSA ]:
38.92000

[ LogP ]:
5.07060

[ 外观性状 ]:
白色细结晶粉末

[ 折射率 ]:
1.612

[ 储存条件 ]:
2-8°C

2-(4-联苯基)-5-苯基恶二唑MSDS

2-(4-联苯基)-5-苯基恶二唑安全信息

[ 个人防护装备 ]:
Eyeshields;Gloves;type N95 (US);type P1 (EN143) respirator filter

[ 危害码 (欧洲) ]:
Xi

[ 安全声明 (欧洲) ]:
S22-S24/25

[ 危险品运输编码 ]:
NONH for all modes of transport

[ WGK德国 ]:
3

[ 海关编码 ]:
2934999090

2-(4-联苯基)-5-苯基恶二唑合成路线

2-(4-联苯基)-5-苯基恶二唑上下游产品

2-(4-联苯基)-5-苯基恶二唑上游产品

2-(4-联苯基)-5-苯基恶二唑下游产品

2-(4-联苯基)-5-苯基恶二唑海关

[ 海关编码 ]: 2934999090

[ 中文概述 ]:
2934999090. 其他杂环化合物. 增值税率:17.0%. 退税率:13.0%. 监管条件:无. 最惠国关税:6.5%. 普通关税:20.0%

[ 申报要素 ]: 品名, 成分含量, 用途

[ Summary ]:
2934999090. other heterocyclic compounds. VAT:17.0%. Tax rebate rate:13.0%. . MFN tariff:6.5%. General tariff:20.0%

2-(4-联苯基)-5-苯基恶二唑文献

Cytohesin 2/ARF6 regulates preadipocyte migration through the activation of ERK1/2.

Biochem. Pharmacol. 92(4) , 651-60, (2015)

Preadipocyte migration is vital for the development of adipose tissue, which plays a crucial role in lipid metabolism. ADP-ribosylation factor 6 (ARF6) small GTPase, which regulates membrane trafficki...

mTOR Directs Breast Morphogenesis through the PKC-alpha-Rac1 Signaling Axis.

PLoS Genet. 11 , e1005291, (2015)

Akt phosphorylation is a major driver of cell survival, motility, and proliferation in development and disease, causing increased interest in upstream regulators of Akt like mTOR complex 2 (mTORC2). W...

Suppression of chemotaxis by SSeCKS via scaffolding of phosphoinositol phosphates and the recruitment of the Cdc42 GEF, Frabin, to the leading edge.

PLoS ONE 9(10) , e111534, (2014)

Chemotaxis is controlled by interactions between receptors, Rho-family GTPases, phosphatidylinositol 3-kinases, and cytoskeleton remodeling proteins. We investigated how the metastasis suppressor, SSe...

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