Cell apoptosis, sometimes called programmed cell death, is a cellular self-destruction method to remove old and damaged cells during development and aging to protect cells from external disturbances and maintain homeostasis. Apoptosis also occurs as a defense mechanism such as in immune reactions or when cells are damaged by disease or noxious agents.

Apoptosis is controlled by many genes and involves two fundamental pathways: the extrinsic pathway, which transmits death signals by the death receptor (DR), and the intrinsic or mitochondrial pathway. The extrinsic apoptotic pathway is activated by the binding of the death ligand to DRs, including FasL, TNF-α, and TRAIL, on the plasma membrane. The DR, adaptor protein (FADD), and associated apoptosis signaling molecule (caspase-8) form the death-inducing signaling complex (DISC), thus leading to the activation of the effector caspase cascade (caspase-3, -6, and -7). The mitochondria-mediated intrinsic apoptosis pathway is regulated by Bcl-2 family proteins, including proapoptotic (Bid, Bax, Bak) and antiapoptotic proteins (Bcl-2, Bcl-xL).

Abnormalities in cell apoptosis can be a significant component of diseases such as cancer, autoimmune lymphoproliferative syndrome, AIDS, ischemia, and neurode-generative diseases. These diseases may benefit from artificially inhibiting or activating apoptosis. A short list of potential methods of anti-apoptotic therapy includes stimulation of the IAP (inhibitors of apoptosis proteins) family of proteins, caspase inhibition, PARP (poly [ADP-ribose] polymerase) inhibition, stimulation of the PKB/Akt (protein kinase B) pathway, and inhibition of Bcl-2 proteins.

Ferroptosis and necroptosis are recently recognized forms of regulated cell death that differs considerably from apoptosis. Misregulated ferroptosis or necroptosis have also been implicated in multiple physiological and pathological processes, including cancer cell death, neurotoxicity, neurodegenerative diseases, etc.

References:
[1] Susan Elmore. Toxicol Pathol. 2007; 35(4): 495–516.
[2] Cao L, et al. J Cell Death. 2016 Dec 29;9:19-29.
[3] Dasgupta A, et al. Int J Mol Sci. 2017 Jan; 18(1): 23.
[4] Xie Y, et al. Cell Death Differ. 2016 Mar;23(3):369-79.


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Dehydrocorydalin

Dehydrocorydaline (13-Methylpalmatine) is an alkaloid isolated from traditional Chinese herb Corydalis yanhusuo W.T. Wang. Dehydrocorydaline regulates protein expression of Bax, Bcl-2; activates caspase-7, caspase-8, and inactivates PARP.

  • CAS Number: 30045-16-0
  • MF: C22H24NO4+
  • MW: 366.43000
  • Catalog: Bcl-2 Family
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: 170-173℃
  • Flash Point: N/A

Artesunate-d4

Artesunate-d4 is deuterium labeled Artesunate. Artesunate is an inhibitor of both STAT-3 and exported protein 1 (EXP1).

  • CAS Number: 1316753-15-7
  • MF: C19H24D4O8
  • MW: 388.45
  • Catalog: Ferroptosis
  • Density: 1.3±0.1 g/cm3
  • Boiling Point: 507.1±50.0 °C at 760 mmHg
  • Melting Point: 130-132°C
  • Flash Point: 175.6±23.6 °C

Minnelide

Minnelide is a prodrug of triptolide that shows potent antitumor activity in a number of tumor types, particularly in pancreatic cancer. Minnelide causes apoptotic[1].

  • CAS Number: 1254702-87-8
  • MF: C21H25Na2O10P
  • MW: 514.37
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

4-Nitroquinoline 1-oxide

Nitrochin (4-NQO) is a chemical carcinogen. Nitrochin induces oncostatin-M (OSM) in esophageal cells. Nitrochin induces DNA damage, and induces apoptosis via a p53-dependent mitochondrial signaling pathway[1][2].

  • CAS Number: 56-57-5
  • MF: C9H6N2O3
  • MW: 190.16
  • Catalog: Apoptosis
  • Density: 1.4±0.1 g/cm3
  • Boiling Point: 387.6±34.0 °C at 760 mmHg
  • Melting Point: 154-156 °C(lit.)
  • Flash Point: 188.2±25.7 °C

Licofelone-d6

Licofelone-d6 is the deuterium labeled Licofelone[1]. Licofelone (ML-3000) is a dual COX/5-lipoxygenase (5-LOX) inhibitor (IC50=0.21/0.18 μM, respectively) for the treatment of osteoarthritis. Licofelone exerts anti-inflammatory and anti-proliferative effects. Licofelone induces apoptosis, and decreases the production of proinflammatory leukotrienes and prostaglandins[2][3][4].

  • CAS Number: 1178549-81-9
  • MF: C23H16D6ClNO2
  • MW: 385.92
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

allethrin

Allethrin, a pyrethroid insecticide is a major mosquito repellent agent. Allethrin induces oxidative stress, apoptosis and calcium release in rat testicular carcinoma cells (LC540). Allethrin induces BCL-2, caspase-3 activation and release of intracellular calcium[1].

  • CAS Number: 584-79-2
  • MF: C19H26O3
  • MW: 302.408
  • Catalog: Apoptosis
  • Density: 1.1±0.1 g/cm3
  • Boiling Point: 386.8±42.0 °C at 760 mmHg
  • Melting Point: 51ºC
  • Flash Point: 166.0±27.9 °C

Carteolol

Carteolol is a non-selective β-adrenoceptor antagonist. Carteolol induces apoptosis via a caspase activated and mitochondrial-dependent pathway. Carteolol can be used for glaucoma research[1].

  • CAS Number: 51781-06-7
  • MF: C16H24N2O3
  • MW: 292.37300
  • Catalog: Bcl-2 Family
  • Density: 1.13 g/cm3
  • Boiling Point: 518.6ºC at 760 mmHg
  • Melting Point: N/A
  • Flash Point: 267.4ºC

A-1293102

A-1293102 is a potent and selective BCL-XL inhibitor that kills BCL-XL-dependent tumor cells[1].

  • CAS Number: 1565759-96-7
  • MF: C42H40F3N7O7S5
  • MW: 972.13
  • Catalog: Bcl-2 Family
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

GPLGIAGQ

GPLGIAGQ, a MMP2-cleavable polypeptide, is used as a stimulus-sensitive linker in both liposomal and micellar nanocarriers for MMP2-triggered tumor targeting. GPLGIAGQ can be used to synthesis unique MMP2-targeted photosensitizer in photodynamic therapy (PDT)[1][2][3].

  • CAS Number: 109053-09-0
  • MF: C31H53N9O10
  • MW: 711.81
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

AMG232

AMG 232 is a potent, selective and orally available inhibitor of p53-MDM2 interaction, with an IC50 of 0.6 nM. AMG 232 binds to MDM2 with a Kd of 0.045 nM.

  • CAS Number: 1352066-68-2
  • MF: C28H35Cl2NO5S
  • MW: 568.55200
  • Catalog: MDM-2/p53
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

BiP inducer X

BiP inducer X, a selective inducer of immunoglobulin heavy chain binding protein (BiP)/GRP78, is an effective ER (endoplasmic reticulum) stress inhibitor. BiP inducer X preferentially induces BiP with slight inductions of GRP94, calreticulin, and C/EBP homologous protein. BiP inducer X protects neurons from ER stress[1][2].

  • CAS Number: 101714-41-4
  • MF: C9H7NO3S
  • MW: 209.22
  • Catalog: Apoptosis
  • Density: 1.5±0.1 g/cm3
  • Boiling Point: 517.1±45.0 °C at 760 mmHg
  • Melting Point: 127 °C
  • Flash Point: 266.5±28.7 °C

c-Met-IN-14

c-Met-IN-14 (compound 26af) is a selective inhibitor of c-Met kinase from N-sulfonylamidine-based derivatives, with an IC50 value of 2.89 nM. c-Met-IN-14 shows anticancer activity by blocking phosphorylation of c-Met, and arrests cell cycle at G2/M phase. c-Met-IN-14 induces apoptosis of A549 cells in a dose-dependent manner[1].

  • CAS Number: 2443380-34-3
  • MF: C34H38ClFN4O7S
  • MW: 701.20
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

Ky1022

KY1022 is a ras destabilizer. KY1022 targets the Wnt/ß-catenin pathway and inhibits development of metastatic colorectal cancer.

  • CAS Number: 1029721-36-5
  • MF: C17H19N3OS
  • MW: 313.42
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

ASR-488

ASR-488 activates the mRNA-binding protein CPEB1, induces apoptosis and inhibits bladder cancer growth[1].

  • CAS Number: 2690312-65-1
  • MF: C33H40O7S
  • MW: 580.73
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

isodispar B

Isodispar B is an anticancer agent that inhibits the proliferation of nasopharyngeal carcinoma and breast cancer cells and induces cell apoptosis. Isodispar B is cytotoxic to a wide range of cancer cell lines[1].

  • CAS Number: 98192-64-4
  • MF: C20H18O5
  • MW: 338.35400
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

RIPK2-IN-8

RIPK-IN-4 is a potent and selective RIPK2 inhibitor with excellent oral bioavailability, and has an IC50 of 3 nM[1].

  • CAS Number: 2141969-56-2
  • MF: C18H21FN4O3S
  • MW: 392.45
  • Catalog: RIP kinase
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

Antiproliferative agent-32

Antiproliferative agent-32 (Compound 1c) inhibits the phosphorylation of PI3K/Akt/mTOR signaling pathway. Antiproliferative agent-32 inhibits Huh7 and SK-Hep-1 cells proliferation, and induce cells apoptosis, causes mitochondrial damage. Antiproliferative agent-32 can be used for research of hepatocellular carcinoma[1].

  • CAS Number: 2925814-05-5
  • MF: C19H15NO2
  • MW: 289.33
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

Zileuton-d4

Zileuton-d4 (A 64077-d4) is the deuterium labeled Zileuton. Zileuton (A 64077) is a potent and selective inhibitor of 5-lipoxygenase with antiasthmatic properties[1][2].

  • CAS Number: 1189878-76-9
  • MF: C11H8D4N2O2S
  • MW: 240.31500
  • Catalog: Ferroptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

Nimustine HCL

Nimustine hydrochloride (ACNU) is a DNA cross-linking and DNA alkylating agent, which induces DNA replication blocking lesions and DNA double-strand breaks and inhibits DNA synthesis, commonly used in chemotherapy for glioblastomas[1][2][3].

  • CAS Number: 55661-38-6
  • MF: C9H14Cl2N6O2
  • MW: 309.152
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: 186 °C
  • Flash Point: N/A

Thiocolchicine-d3

Thiocolchicine-d3 is deuterium labeled Thiocolchicine. Thiocolchicine, a derivative modified in the C Ring of Colchicine (HY-16569) with enhanced biological properties. Thiocolchicine is a potent inhibitor of tubulin polymerization (IC50=2.5 µM) and competitively binds to tubulin with a Ki of 0.7 µM. Thiocolchicine induces cell apoptosis[1][2]. Thiocolchicine can be used as an ADC cytotoxin in ADC technology.

  • CAS Number: 1314417-95-2
  • MF: C22H22D3NO5S
  • MW: 418.52
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

Cu(II)-Elesclomol

Cu(II)-Elesclomol is a Cu2+ complex of Elesclomol (HY-12040). Cu(II)-Elesclomol induces apoptosis, causes a G1 cell cycle block and induces DNA double strand breaks in K562 cells. Cu(II)-Elesclomol also weakly inhibits DNA topoisomerase I. Cu(II)-Elesclomol has anticancer activity[1].

  • CAS Number: 1224195-72-5
  • MF: C19H18CuN4O2S2
  • MW: 462.05
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

Myristoleic acid

Myristoleic acid, a cytotoxic component in the extract from Serenoa repens, induces apoptosis and necrosis in human prostatic LNCaP cells[1].

  • CAS Number: 544-64-9
  • MF: C14H26O2
  • MW: 226.355
  • Catalog: Apoptosis
  • Density: 0.9±0.1 g/cm3
  • Boiling Point: 338.9±0.0 °C at 760 mmHg
  • Melting Point: -4.5--4ºC(lit.)
  • Flash Point: 206.5±14.4 °C

MI-192 HCl

MI-192 is a selective HDAC2 and HDAC3 inhibitor with IC50s of 30 nM and 16 nM, respectively. MI-192 is more selective for HDAC2/3 than other HDAC isomers.MI-192 induces myeloid leukaemic cells apoptosis. Anticaner and neuroprotective activities[1][2].

  • CAS Number: 1415340-63-4
  • MF: C24H21N3O2
  • MW: 383.44
  • Catalog: Apoptosis
  • Density: 1.3±0.1 g/cm3
  • Boiling Point: 565.0±50.0 °C at 760 mmHg
  • Melting Point: N/A
  • Flash Point: 295.5±30.1 °C

Mcl1-IN-9

Mcl1-IN-9 is a potent myeloid cell leukemia-1 (Mcl-1) Inhibitor with an IC50 of 446 nM in reengineered BCR-ABL+ B-ALL cells and a Ki of 0.03 nM[1].

  • CAS Number: 1810769-31-3
  • MF: C37H39ClN4O4
  • MW: 639.18
  • Catalog: Bcl-2 Family
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

Z-IETD-FMK

Z-IETD-FMK is a selective and cell permeable caspase 8 inhibitor.

  • CAS Number: 210344-98-2
  • MF: C30H43FN4O11
  • MW: 654.681
  • Catalog: Peptides
  • Density: 1.2±0.1 g/cm3
  • Boiling Point: 925.7±65.0 °C at 760 mmHg
  • Melting Point: N/A
  • Flash Point: 513.6±34.3 °C

PTC596

PTC596 is an orally active and selective B-cell-specific Moloney murine leukemia virus integration site 1 (BMI-1) inhibitor. PTC596 targets BMI1 expressed by both tumor cells and cancer stem cells (CSCs), and induces hyper-phosphorylation of BMI1, leading to its degradation. PTC596 downregulates MCL-1 and induces p53-independent mitochondrial apoptosis in acute myeloid leukemia progenitor cells[1][2].

  • CAS Number: 1610964-64-1
  • MF: C19H13F5N6
  • MW: 420.339
  • Catalog: Apoptosis
  • Density: 1.6±0.1 g/cm3
  • Boiling Point: 583.9±60.0 °C at 760 mmHg
  • Melting Point: N/A
  • Flash Point: 307.0±32.9 °C

Epipodophyllotoxin

(-)-Epipodophyllotoxin (2) is an antiproliferative agent against cancer cells isolated from American mayapple Podophyllum peltatum, with GI50s of 0.36 and 0.24 μM in HeLa cells and MCF-7 cells, respectively. (-)-Epipodophyllotoxin can inhibit mitotic spindle assembly in vitro[1].

  • CAS Number: 4375-07-9
  • MF: C22H22O8
  • MW: 414.40500
  • Catalog: Apoptosis
  • Density: 1.37g/cm3
  • Boiling Point: 597.9ºC at 760 mmHg
  • Melting Point: 160.3 °C
  • Flash Point: 210.2ºC

Aristololactam I

Aristololactam I (AL-I), is the main metabolite of aristolochic acid I (AA-I), participates in the processes that lead to renal damage.Aristololactam I (AL-I) directly injures renal proximal tubule cells, the cytotoxic potency of AL-I is higher than that of AA-I and that the cytotoxic effects of these molecules are mediated through the induction of apoptosis in a caspase-3-dependent pathway[1].

  • CAS Number: 13395-02-3
  • MF: C17H11NO4
  • MW: 293.273
  • Catalog: Caspase
  • Density: 1.5±0.1 g/cm3
  • Boiling Point: 463.9±34.0 °C at 760 mmHg
  • Melting Point: 319 °C
  • Flash Point: 234.4±25.7 °C

TS-IN-2

TS-IN-2 (compound 17) is a thymidylate synthase (TS) inhibitor with an IC50 of 0.81 μM. TS-IN-2 induces cancer cell apoptosis and cell cycle arrest in S phase, and also inhibit DNA synthesis, resulting in DNA damage[1].

  • CAS Number: 2945983-00-4
  • MF: C18H23N3O4S
  • MW: 377.46
  • Catalog: Thymidylate Synthase
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

g-Keto-b-methoxy-d-methylene-Da-hexenoic Acid

Penicillic acid is a polyketide mycotoxin produced by several species of Aspergillus and Penicillium, which exhibits cytotoxicity in rat alveolar macrophages (AM) in vitro. Penicillic acid inhibits Fas ligand-induced apoptosis by blocking self-processing of caspase-8[1][2].

  • CAS Number: 90-65-3
  • MF: C8H10O4
  • MW: 170.163
  • Catalog: Caspase
  • Density: 1.2±0.1 g/cm3
  • Boiling Point: 285.7±35.0 °C at 760 mmHg
  • Melting Point: 83-87ºC
  • Flash Point: 113.5±19.4 °C